ACTION OF ALCOHOL ON INTERNAL ORGANS.
ACTION OF ALCOHOL ON INTERNAL ORGANS. |
Alcohol's harmful effects on the stomach make it impossible for it to absorb food, which could lead to incomplete digestion, and prevent it from producing enough of its own digestive juices. An alcoholic will always experience nausea, a feeling of emptiness, distention, and prostration. This causes a dislike of eating and is accompanied by a desire for more alcohol. This leads to the development of a chronic illness known as dyspepsia. This procedure is the source of confirmed cases of devastating dyspepsia.
How this impacts the liver.
Persistent alcohol consumption can lead to organic deteriorations that can be deadly. The liver is the organ that is most commonly affected structurally by alcohol. The liver's cellular components can typically store active chemicals. In cases of poisoning caused by different toxic substances, we examine the liver as though it were the main storage location for the foreign material. With regard to alcohol, it is much the same. An alcoholic's liver is too frequently saturated with alcohol and is never truly free from its impact. The liver's tiny membranous or capsular structure is impacted, which hinders appropriate dialysis and unrestricted secretion. The enlargement of the liver is caused by the dilation of its arteries, the accumulation of fluid, and the thickening of its tissue. This is the result of the membrane contracting and the entire organ's cellular components shrinking. The obstruction that the veins provide to the returning blood causes the alcoholic's lower body to become dropsical. It is possible for the liver's structure to become charged with fatty cells and experience what is known as "fatty liver."
The way the kidneys fail.
Drinking too much alcohol has a negative impact on the kidneys as well. The kidneys' blood arteries lose their flexibility and contraction force. Their microscopic structures undergo lipid change. Their membranes are easily penetrated by blood albumin. The body progressively loses power as a result, as if it were gradually running out of blood.
obstruction of the airways.
Because the lungs' blood arteries are most exposed to temperature changes, alcohol readily relaxes them. They become easily clogged when exposed to the impacts of a sudden change in air temperature. An alcoholic is easily affected by the unexpectedly deadly congestions of the lungs during harsh winter seasons.
Alcohol causes cardiac weakness.
This is
Alcohol use has a significant negative impact on the heart. The thicker, more cartilaginous or calcareous membraneous tissues that surround and border the heart undergo changes in quality. Subsequently, the valves become less flexible, leading to a persistent condition known as valvular dysfunction. The big blood artery that emerges from the heart has structural changes in its coats, which results in the vessel losing its elasticity and ability to supply the heart when it recoils from its distention after the heart has filled it with blood through a stroke.
Once more, the heart's muscular framework collapses as a result of tissue deterioration. Fatty cells either replace the components of the muscle fiber or, if they are not, they themselves are translated into a modified muscular texture with a significantly decreased contraction power.
People who experience these organic deteriorations of the main organ that controls blood circulation discover this reality so gradually that it doesn't really hit them until the damage has already done significant damage. They recognize a central breakdown in their power resulting from minor factors like fatigue, conflict, interrupted sleep, or prolonged fasting. They describe it as a "sinking," yet they are aware that wine or some stimulant will instantly make it go away. So they try to alleviate it till they finally find that the solution doesn't work. The weary, exhausted, and devoted heart can take no more. The bloodstream's governor has been shattered and it has served its purpose. Either the current eventually spills into the tissues damming up the courses or completely stopping in the center after a small shock or excess motion.